I have training in biochemistry and obtained a Ph.D. in Cell and Molecular Biology. I have expertise in neurobiology and neurodegenerative diseases. Since my doctoral work, I became interested in the molecular mechanisms involved in neurodegenerative diseases. In my Thesis, I characterized the effect of Acetylcholinesterase promoting the Aβ peptide aggregation. In my post-doctorate, I studied the signal transduction pathways involved in neuronal cytoskeleton alterations, tau phosphorylation, and neuronal death in Alzheimer's disease (AD). In the last years, I have been actively conducting research focused on understanding the role of c-Abl tyrosine kinase signaling in Neurodegenerative diseases. Our work has shown that c-Abl is activated in both in vitro and in vivo AD models and mediates neuronal death, tau phosphorylation, and cognitive impairments. Also, we demonstrated that AβO binds to Ephrin receptor A4 causing c-Abl activation and the downstream synaptic loss, LTP blockade, and gene repression of synaptic genes. Our recent results support the role of dysregulated c-Abl signaling in neurons damage and seizures induction in temporal lobe epilepsy. I have formed several undergraduate students, Ph. D. students, and post-doctorates, so I have experience in human resources training.