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Introduction
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Contribution of monocytes and CD4 T cell subsets in maintaining viral reservoirs in SIV-infected macaques treated early after infection with antiretroviral drugs
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Cell proliferation contributes to the increase of genetically intact HIV over time
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Multiple sanctuary sites for intact and "defective" HIV-1 in post-mortem tissues in individuals with suppressed HIV-1 replication: Implications for HIV-1 cure strategies
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High-throughput characterization of HIV latent reservoir demonstrates integration into genes associated with inflammation, cell cycle, and nuclear envelope assembly, enrichment in accessible chromatin, and large amounts of defective provirus
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The size of HIV reservoir is associated with telomere shortening and immunosenescence in early ART-treated HIV-infected children
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Conclusion
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