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Introduction
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SMAC mimetic plus triple combination bispecific HIVxCD3 DART® molecules in SHIV.CH505-infected, ART-suppressed rhesus macaques
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Infection outcome in RT-SHIV infected macaques treated early with antiretroviral therapy alone or in combination with the TLR7 agonist vesatolimod
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Tyrosine kinase inhibitors promote antiviral resistance in CD4+ T cells against HIV-1 infection even after treatment withdrawal
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Combining a conditional suicide gene with CCR5 knockout for anti-HIV gene therapy
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Gene therapy with an anti-tat gene can strongly block HIV-1 transcription and virus replication in mouse models of infection
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Conclusion
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