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A01.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding

P002 - THE ROLE OF CELL STRESS IN DRIVING PROTEIN AGGREGATION WITHIN CONTROL IPSC-DERIVED NEURONS (ID 1261)

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P004 - ANALYSIS OF BETA-AMYLOID CYTOTOXICITY AS A FUNCTION OF STRUCTURE AND AGGREGATION CONDITIONS (ID 1700)

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P006 - SYNTHETIC AND NATURAL CONFORMATIONAL VARIANTS OF MISFOLDED AΒ INDUCE DIFFERENT PATHOLOGICAL FEATURES IN SUSCEPTIBLE MICE. (ID 1742)

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P008 - DEFICIENT COGNITIVE AND EMOTIONAL FUNCTIONS AND NEUROTRANSMITTER LEVELS IN THE TGDIMER MOUSE THAT EXPRESSES AMYLOID-BETA DIMERS, BUT NO PLAQUE PATHOLOGY (ID 2152)

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P009 - AN IN VITRO PLATFORM FOR MODELING AND MEASURING THE OLIGOMER STATE IN THE AMYLOID Β GROWTH DYNAMICS (ID 2212)

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P010 - ROLE OF PRO-INFLAMMATORY S100 PROTEINS IN AMYLOID NEUROINFLAMMATORY CASCADE IN NEURODEGENERATIVE DISEASES. (ID 226)

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P011 - THE MODE OF SYNERGISTIC SUPPRESSION ON LARGE CONDUCTANCE CALCIUM-ACTIVATED POTASSIUM CHANNEL BY AMYLOID BETA OLIGOMER AND AMYLOID PRECURSOR PROTEIN (ID 281)

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P012 - MODELING ALZHEIMER DISEASE-DOWN SYNDROME IN THE FLY (ID 305)

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P013 - THE UPDATED "UNIFYING HYPOTHESIS OF ALZHEIMER'S DISEASE" - HEPARAN SULFATE PROTEOGLYCANS/ GLYCOSAMINOGLYCANS ARE KEY AS FIRST HYPOTHESIZED OVER 30 YEARS AGO (ID 701)

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P014 - HUMAN AMYLOID SEEDS AGGREGATE MORE EFFICIENT THAN SEEDS FROM OLD 3XTG-AD MICE (ID 785)

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P015 - ASSOCIATION OF AMYLOID Β AND DNA REPAIR ACTIVITY OF MIDDLE-AGED WOMEN PARTICIPATED IN BEACH FITNESS RETREAT PROGRAM (ID 823)

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P017 - MEMBRANE COMPOSITION CAN MODULATE CELLULAR RESILIENCY TO AMYLOID-BETA42 OLIGOMER TOXICITY (ID 983)

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