Theme A: β-Amyloid Diseases || A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
ePoster not uploaded
P015 - MELATONIN INHIBIT THE APOPTOSIS BY JNK INHIBTION THROUGH IMPROVED MITOCHONDRIAL FUNCTION IN AD
Presentation Topic
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
Presenter
Authors
P016 - PRODUCTION OF AΒ HEXAMERS IN AD PATHOLOGY
Presentation Topic
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
Presenter
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
ePoster not uploaded
P017 - EXTRACELLULAR VESICLES ARE ENRICHED IN OLIGOMERIC APP-CTFS IN AD MODELS
Presentation Topic
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
Presenter
P018 - BRAIN EXPRESSION AND PROCESSING OF THE AMYLOID PRECURSOR PROTEIN IS UNAFFECTED BY APOLIPOPROTEIN E GENOTYPE
Presentation Topic
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
Presenter
P019 - TRIFUNCTIONAL NANOPARTICLE DESIGN TO RESCUE NEUROSYSTEM FAILURE CAUSED BY PROTEIN AGGREGATION (TRINADES)
Presentation Topic
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
Presenter
P020 - SULFONYLUREA RECEPTOR/SUR DEFICIENCY INCREASED VULNERABILITY TO NEURODEGENERATION IN DROSOPHILA MODELS OF ALZHEIMER’S DISEASE
Presentation Topic
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
Presenter
P021 - DEPLETION OF CAVEOLIN-1 IN TYPE-2 DIABETES INDUCES ALZHEIMER’S DISEASE PATHOLOGY
Presentation Topic
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
ePoster not uploaded
P022 - AMYLOID-B SEEDING IN A SPORADIC MODEL OF ALZHEIMER’S DISEASE
Presentation Topic
A1.a. Disease Mechanisms, Pathophysiology: Abeta aggregation, protein misfolding
Presenter
Authors
- Laura Trujillo-Estrada, Spain
- Kelly Do Huynh, United States of America
- Marie Minh Thu Nguyen, United States of America
- Alwin Cheung, United States of America
- Janine Pham Tran, United States of America
- Cristina Nuñez-Diaz, Spain
- Stefania Forner, United States of America
- Alessandra Martini, United States of America
- Celia Da Cunha, United States of America
- Mohammad Shahnawaz, United States of America
- Claudio Soto, United States of America
- Ines Moreno Gonzalez, Spain
- Antonia Gutierrez, Spain
- Frank Laferla, United States of America
- David Baglietto-Vargas, Spain
Theme A: β-Amyloid Diseases || A1.b. Disease Mechanisms, Pathophysiology: Cell to cell transmission, spreading of pathology, prion-like
A1.b. Disease Mechanisms, Pathophysiology: Cell to cell transmission, spreading of pathology, prion-like
P024 - THE SPEED LIMITS OF TAU SPREADING - THE CONTRIBUTION OF EDUCATION AND REGIONAL AMYLOID
Presentation Topic
A1.b. Disease Mechanisms, Pathophysiology: Cell to cell transmission, spreading of pathology, prion-like
Presenter
A1.b. Disease Mechanisms, Pathophysiology: Cell to cell transmission, spreading of pathology, prion-like
P025 - THE ROLE OF THE RETROMER, AUTOPHAGY AND EXOSOMES IN THE CLEARANCE AND PROPAGATION OF AMYLOID BETA OLIGOMERS IN ALZHEIMER’S DISEASE
Presentation Topic
A1.b. Disease Mechanisms, Pathophysiology: Cell to cell transmission, spreading of pathology, prion-like
Presenter
A1.b. Disease Mechanisms, Pathophysiology: Cell to cell transmission, spreading of pathology, prion-like
P026 - SOLUBLE HIGH-MOLECULAR-WEIGHT AΒ OLIGOMERS DERIVED FROM THE AΒ-LADEN BRAINS INDUCE CEREBRAL Β-AMYLOIDOSIS.
Presentation Topic
A1.b. Disease Mechanisms, Pathophysiology: Cell to cell transmission, spreading of pathology, prion-like
Presenter
A1.b. Disease Mechanisms, Pathophysiology: Cell to cell transmission, spreading of pathology, prion-like
P027 - EVALUATION OF ALZHEIMER’S DISEASE HETEROGENEITY THROUGH HUMAN BRAIN INOCULATIONS IN A MOUSE MODEL OF AMYLOID AND TAU LESIONS
Presentation Topic
A1.b. Disease Mechanisms, Pathophysiology: Cell to cell transmission, spreading of pathology, prion-like
Presenter